Looks like its about time certain things come to an end. For instance, our latest world shocking epidemic, Type A H1N1 a.k.a Swine Flu a.k.a Selesema BABI!

Yeah, I ain't kidding you....

Well, lets see....first of all you gotta get a hold of this thing called Hydroxytyrosol(HT), its an inactivated influenza type A viruses. It has an antiviral effect as long as the viral envelope (H1N1 virus) is present in your body.

Simple to say that, if you have swine flu, get this HT into your body, and since its weakened, your body will be able to make your antibodies stronger by killing these viruses to actually fight the "BIG ONE" that is in your system. Let us all give the Japanese a round of applause again for their advance technology and brains in this field. *clap*clap*clap*

Here is the clipped abstract of their research:

Mechanism of the antiviral effect of hydroxytyrosol on influenza virus appears to involve morphological change of the virus.

Yamada K, Ogawa H, Hara A, Yoshida Y, Yonezawa Y, Karibe K, Nghia VB, Yoshimura H, Yamamoto Y, Yamada M, Nakamura K, Imai K.

Research Center for Animal Hygiene and Food Safety, Obihiro University of Agriculture and Veterinary Medicine, Obihiro, Hokkaido, Japan.

Hydroxytyrosol (HT), a small-molecule phenolic compound, inactivated influenza A viruses including H1N1, H3N2, H5N1, and H9N2 subtypes. HT also inactivated Newcastle disease virus but not bovine rotavirus, and fowl adenovirus, suggesting that the mechanism of the antiviral effect of HT might require the presence of a viral envelope. Pretreatment of MDCK cells with HT did not affect the propagation of H9N2 virus subsequently inoculated onto the cells, implying that HT targets the virus but not the host cell. H9N2 virus inactivated with HT retained unaltered hemagglutinating activity and bound to MDCK cells in a manner similar to untreated virus. Neuraminidase activity in the HT-treated virus also remained unchanged. However, in the cells inoculated with HT-inactivated H9N2 virus, neither viral mRNA nor viral protein was detected. Electron microscopic analysis revealed morphological abnormalities in the HT-treated H9N2 virus. Most structures found in the HT-treated virus were atypical of influenza virions, and localization of hemagglutinin was not necessarily confined on the virion surface. These observations suggest that the structure of H9N2 virus could be disrupted by HT.

You can follow the original blog link where I got my info from and the research abstract:

OLIVENOL Livin'

”Antiviral Research. 2009 Jul;83(1):35-44”

So, everyone out there who has it, go and get its done before all of us get it!

Peace out y'all, till next time~~